Neuron‐specific translational control shift ensures proteostatic resilience during ER stress | The EMBO Journal
Differential impact of imipramine on thapsigargin- and tunicamycin-induced endoplasmic reticulum stress and mitochondrial dysfunction in neuroblastoma SH-SY5Y cells - ScienceDirect
EGF-SubA enhances cytotoxic effects of the ER stress inducer... | Download Scientific Diagram
ER stress-induced cell death mechanisms - ScienceDirect
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences
Calcium Influx Caused by ER Stress Inducers Enhances Oncolytic Adenovirus Enadenotucirev Replication and Killing through PKCα Activation: Molecular Therapy - Oncolytics
Revealing Protein Aggregates under Thapsigargin-Induced ER Stress Using an ER-Targeted Thioflavin | ACS Sensors
IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to Anticancer Drug
Endoplasmic reticulum stress, the unfolded protein response and autophagy in kidney diseases | Nature Reviews Nephrology
JNK activation by thapsigargin versus saturated free fatty acid (SFA).... | Download Scientific Diagram
Figure S2. Thapsigargin-induced ER Ca 2+ release is reduced in STIM1KO... | Download Scientific Diagram
Thapsigargin - Wikipedia
MITOL prevents ER stress‐induced apoptosis by IRE1α ubiquitylation at ER–mitochondria contact sites | The EMBO Journal
Endoplasmic Reticulum Stress Mediates Vascular Smooth Muscle Cell Calcification via Increased Release of Grp78 (Glucose-Regulated Protein, 78 kDa)-Loaded Extracellular Vesicles | Arteriosclerosis, Thrombosis, and Vascular Biology
Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia
HRC promotes anoikis resistance and metastasis by suppressing endoplasmic reticulum stress in hepatocellular carcinoma
Phenotypic Discovery of Neuroprotective Agents by Regulation of Tau Proteostasis via Stress‐Responsive Activation of PERK Signaling - Shin - 2021 - Angewandte Chemie International Edition - Wiley Online Library
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text
Thapsigargin-induced severe ER stress stimulates release of... | Download Scientific Diagram
Frontiers | STING, the Endoplasmic Reticulum, and Mitochondria: Is Three a Crowd or a Conversation?
Biomedicines | Free Full-Text | The Crosstalk of Endoplasmic Reticulum (ER) Stress Pathways with NF-κB: Complex Mechanisms Relevant for Cancer, Inflammation and Infection
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences
ER stress-induced cell death proceeds independently of the TRAIL-R2 signaling axis in pancreatic β cells | Cell Death Discovery
