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Lipopolysaccharides of Fusobacterium nucleatum and Porphyromonas gingivalis increase RANKL-expressing neutrophils in air pouches of mice | Laboratory Animal Research | Full Text
Visfatin regulates Pg LPS-induced proinflammatory/prodegradative effects in healthy and inflammatory periodontal cells partially via NF-κB pathway - ScienceDirect
GI Bacteria Affects Rheumatoid Arthritis Disease Markers | Chondrex, Inc.
Chlorogenic Acid as a Positive Regulator in LPS-PG-Induced Inflammation via TLR4/MyD88-Mediated NF-κB and PI3K/MAPK Signaling Cascades in Human Gingival Fibroblasts
Hemin-Dependent Modulation of the Lipid A Structure of Porphyromonas gingivalis Lipopolysaccharide | Infection and Immunity
P.g.-infection and LPS-PG induce myofibroblastic activation of HSCs.... | Download Scientific Diagram
Role of TLR4 signaling on Porphyromonas gingivalis LPS-induced cardiac dysfunction in mice | PLOS ONE
Circular RNA PPP1CC promotes Porphyromonas gingivalis-lipopolysaccharide-induced pyroptosis of vascular smooth muscle cells by activating the HMGB1/TLR9/AIM2 pathway - Jie Liu, Yong Wang, Yaoyun Liao, Ying Zhou, Jijin Zhu, 2021
Porphyromonas gingivalis Stimulates Bone Resorption by Enhancing RANKL (Receptor Activator of NF-κB Ligand) through Activatio
Lipoxin A4 promotes autophagy and inhibits overactivation of macrophage inflammasome activity induced by Pg LPS - Jie Zhao, Wenjing Geng, Kefei Wan, Kailei Guo, Fengjun Xi, Xiangqun Xu, Xiujuan Xiong, Xu Huang,
Porphyromonas gingivalis lipopolysaccharides act exclusively through TLR4 with a resilience between mouse and human | Scientific Reports
Porphyromonas gingivalis lipopolysaccharides act exclusively through TLR4 with a resilience between mouse and human | Scientific Reports
em>Porphyromonas gingivalis</em> lipopolysaccharide enhances the proliferation of human periodontal ligament cells via upregulation of cyclin D1, cyclin A and cyclin B1
Inflammatory signals in human oral keratinocytes triggered by P.... | Download Scientific Diagram
Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]
Pg ant its LPS increase ECs death.
Porphyromonas gingivalis Outer Membrane Vesicles as the Major Driver of and Explanation for Neuropathogenesis, the Cholinergic Hypothesis, Iron Dyshomeostasis, and Salivary Lactoferrin in Alzheimer's Disease - IOS Press
The Effect of Porphyromonas gingivalis Lipopolysaccharide on the Pyroptosis of Gingival Fibroblasts | SpringerLink
Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]
LPS-PG | Lipopolysaccharide from Porphyromonas Gingivalis
Frontiers | Pathogenesis of Important Virulence Factors of Porphyromonas gingivalis via Toll-Like Receptors
Porphyromonas gingivalis lipopolysaccharide-induced macrophages modulate proliferation and invasion of head and neck cancer cell lines - ScienceDirect
IJMS | Free Full-Text | Porphyromonas gingivalis Lipopolysaccharides Promote Proliferation and Migration of Human Vascular Smooth Muscle Cells through the MAPK/TLR4 Pathway
LPS-PG | Lipopolysaccharide from Porphyromonas Gingivalis